Search results for "Skin repair"

showing 9 items of 9 documents

Do nonmelanoma skin cancers develop from extra-cutaneous stem cells?

2008

A hypothesis is presented that nonmelanoma skin cancers can develop from extra-cutaneous stem cells, and not exclusively from skin keratinocytes. This idea is supported by recent findings regarding the initiation of cancers in the digestive tract, and by a cancer stem cell model of a neoplasia. It is known that multipotent adult progenitor cells can trans-differentiate into very diverse cellular lineages and can be recruited to areas of profound tissue injury. In these settings, they might also initiate malignant transformation. Some epidemiological data and recent findings regarding mechanisms of wound healing indicate that skin cancers could also originate from bone marrow-derived or othe…

KeratinocytesCancer ResearchPathologymedicine.medical_specialtySkin NeoplasmsBone Marrow CellsCancer stem cellepidermisAnimalsHumansMedicineProgenitor cellSkin repairintegumentary systembusiness.industryStem Cellsmedicine.diseasehematopoietic stem cellsCell Transformation Neoplasticmedicine.anatomical_structureOncologyBone marrowSkin cancerStem cellbusinessKeratinocyteWound healingInternational Journal of Cancer
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A Transgenic Mouse Model of Inducible Macrophage Depletion

2009

Whether the wound macrophage is a key regulatory inflammatory cell type in skin repair has been a matter of debate. A transgenic mouse model mediating inducible macrophage depletion during skin repair has not been used to date to address this question. Here, we specifically rendered the monocyte/macrophage leukocyte lineage sensitive to diphtheria toxin by expressing the lysozyme M promoter-driven, Cre-mediated excision of a transcriptional STOP cassette from the simian DT receptor gene in mice (lysM-Cre/DTR). Application of diphtheria toxin to lysM-Cre/DTR mice led to a rapid reduction in both skin tissue and wound macrophage numbers at sites of injury. Macrophage-depleted mice revealed a …

Genetically modified mouseDiphtheria toxinSkin repairintegumentary systemMonocyteInflammationTransforming growth factor betaBiologyMolecular biologyPathology and Forensic MedicineCell biologymedicine.anatomical_structuremedicinebiology.proteinmedicine.symptomWound healingMyofibroblastThe American Journal of Pathology
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Genetic ablation of mast cells redefines the role of mast cells in skin wound healing and bleomycin-induced fibrosis.

2014

Conclusive evidence for the impact of mast cells (MCs) in skin repair is still lacking. Studies in mice examining the role of MC function in the physiology and pathology of skin regenerative processes have obtained contradictory results. To clarify the specific role of MCs in regenerative conditions, here we used a recently developed genetic mouse model that allows conditional MC ablation to examine MC-specific functions in skin. This mouse model is based on the cell type–specific expression of Cre recombinase in connective tissue–type MCs under control of the Mcpt5 promoter and the Cre-inducible diphtheria toxin receptor–mediated cell lineage ablation by diphtheria toxin. In response to ex…

KeratinocytesPathologymedicine.medical_specialtymedicine.medical_treatmentCellCre recombinaseMice TransgenicDermatologyBiologyBleomycinBiochemistrySkin Diseaseschemistry.chemical_compoundBleomycinMiceFibrosismedicineLeukocytesAnimalsMast CellsMolecular BiologyDiphtheria toxinSkin repairWound HealingAntibiotics AntineoplasticGranulation tissueCell BiologyAblationmedicine.diseaseFibrosisDisease Models Animalmedicine.anatomical_structurechemistryGranulation TissueThe Journal of investigative dermatology
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Differential Roles of Macrophages in Diverse Phases of Skin Repair

2010

Abstract Influx of macrophages plays a crucial role in tissue repair. However, the precise function of macrophages during the healing response has remained a subject of debate due to their functional dichotomy as effectors of both tissue injury and repair. We tested the hypothesis that macrophages recruited during the diverse phases of skin repair after mechanical injury exert specific functions to restore tissue integrity. For this purpose, we developed a mouse model that allows conditional depletion of macrophages during the sequential stages of the repair response. Depletion of macrophages restricted to the early stage of the repair response (inflammatory phase) significantly reduced the…

TransgeneImmunologyMice TransgenicCell SeparationBiologyFlow cytometryMiceSkin Physiological PhenomenamedicineAnimalsImmunology and AllergySkinSkin repairWound HealingSkin Physiological Phenomenamedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionEffectorMacrophagesGranulation tissueFlow CytometryImmunohistochemistryCell biologyMice Inbred C57BLmedicine.anatomical_structureImmunologyImmunohistochemistryFunction (biology)The Journal of Immunology
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5-HT1A Receptor Function Makes Wound Healing a Happier Process

2018

Skin wound healing is a multistage phenomenon that is regulated by cell–cell interplay and various factors. Endogenous serotonin is an important neurotransmitter and cytokine. Its interaction with the serotonin 1A receptor (5-HTR1A) delivers downstream cellular effects. The role of serotonin (5-hydroxytryptamine, 5-HT) and the 5-HT1A receptor has been established in the regeneration of tissues such as the liver and spinal motor neurons, prompting the investigation of the role of 5-HT1A receptor in skin healing. This study assessed the role of 5-HT1A receptor in excisional wound healing by employing an excisional punch biopsy model on 5-Ht1a receptor knockout mice. Post-harvest analysis reve…

0301 basic medicineAgonistmedicine.drug_classmedicine.medical_treatmentwound healingPharmacology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineskin regenerationmedicine5-HT1A receptorPharmacology (medical)NeurotransmitterReceptor5-Ht1a receptor knockout mice modelOriginal ResearchPharmacologySkin repairintegumentary systembusiness.industrylcsh:RM1-950serotonin3. Good healthlcsh:Therapeutics. Pharmacology030104 developmental biologyCytokinenervous systemchemistry030220 oncology & carcinogenesisKnockout mouse5-HT1A receptorWound healingbusinessFrontiers in Pharmacology
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Opposite Effects of Mechanical Action of Fluid Flow on Proangiogenic Factor Secretion From Human Adipose-Derived Stem Cells With and Without Oxidativ…

2017

Mechanical forces, hypoxia, and oxidative stress contribute to skin renewal, perfusion, and wound healing, but how are they regulating subcutaneous adipose-derived stem cells (ASCs) in the inflammatory microenvironment associated to skin repair and disorders is unknown. In this study, ASCs were isolated from lipoaspirate samples from plastic surgery patients, primary cultured and their differentiation and secretion of a panel of cytokines with pronounced effects on skin repair and angiogenesis were studied under mechanical stimulation by intermittent fluid flow, 1% hypoxia and oxidative stress by glutathione (GSH) depletion with buthionine sulfoximine (BSO) treatment. Mechanical action of f…

0301 basic medicineSkin repairmedicine.medical_specialtyPhysiologyAngiogenesisClinical BiochemistryAdipose tissueCell BiologyBiologymedicine.disease_cause03 medical and health scienceschemistry.chemical_compound030104 developmental biology0302 clinical medicineEndocrinologychemistry030220 oncology & carcinogenesisInternal medicinemedicineButhionine sulfoximineHuman umbilical vein endothelial cellStem cellWound healingOxidative stressJournal of Cellular Physiology
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Nanodesign of new self-assembling core-shell gellan-transfersomes loading baicalin and in vivo evaluation of repair response in skin

2017

Gellan nanohydrogel and phospholipid vesicles were combined to incorporate baicalin in new self-assembling core-shell gellan-transfersomes obtained by an easy, scalable method. The vesicles were small in size (~107 nm) and monodispersed (P.I. ≤ 0.24), forming a viscous system (~24 mPa/s) as compared to transfersomes (~1.6 mPa/s), as confirmed by rheological studies. Gellan was anchored to the bilayer domains through cholesterol, and the polymer chains were distributed onto the outer surface of the bilayer, thus forming a core-shell structure, as suggested by SAXS analyses. The optimal carrier ability of core-shell gellan-transfersomes was established by the high deposition of baicalin in th…

3003SwinePharmaceutical ScienceMedicine (miscellaneous)02 engineering and technology01 natural sciencesMicechemistry.chemical_compoundDrug Delivery Systemsmaterials science (all)skin deliveryGeneral Materials ScienceSkinchemistry.chemical_classificationSkin repairSmall-angle X-ray scatteringBilayerVesicleAnti-Inflammatory Agents Non-SteroidalPolysaccharides BacterialPolymer021001 nanoscience & nanotechnologymedicine.anatomical_structureMolecular MedicineFemale0210 nano-technologytransfersomesSkin AbsorptionBiomedical EngineeringgellanBioengineeringAdministration Cutaneous010402 general chemistryIn vivo studiesDermisIn vivoSAXS analysismedicineAnimalsgellan; In vivo studies; rheological studies; SAXS analysis; skin delivery; transfersomes; bioengineering; medicine (miscellaneous); molecular medicine; biomedical engineering; materials science (all); 3003rheological studiesFlavonoidsInflammationWound Healing0104 chemical sciencesAnimals NewbornchemistryLiposomesBiophysicsNanoparticlesBaicalin
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Enhancement of lysine acetylation accelerates wound repair

2013

In physiopathological conditions, such as diabetes, wound healing is significantly compromised and chronic complications, including ulcers, may occur. In a mouse model of skin repair, we recently reported that wound treatment with Sirtuin activators and class I HDAC inhibitors induced keratinocyte proliferation and enhanced healing via a nitric oxide (NO) dependent mechanism. We observed an increase in total protein acetylation in the wound area, as determined by acetylation of α-tubulin and histone H3 Lysine 9. We reasoned that this process activated cell function as well as regulated gene expression to foster tissue repair. We report here that the direct activation of P300/CBP-associated …

Skin repairWound HealingepigeneticsActivator (genetics)Short CommunicationkeratinocyteBiologyNitric OxideCell biologyHistone H3medicine.anatomical_structureepigenetics; pathology Wound Healing; lysine acetylation; PCAF; keratinocyte; Nitric OxideBiochemistryPCAFAcetylationPCAFSirtuinmedicinebiology.proteinpathologyGeneral Agricultural and Biological SciencesKeratinocyteWound healinglysine acetylationCommunicative & Integrative Biology
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The Role of Exosomes Derived From Mesenchymal Stromal Cells in Dermatology

2021

This study has been funded by the Carlos III Health Institute of Spain through the PI13/02576 and PI17/02083 projects [cofunded by European Regional Development Fund "A way to make Europe" and Andalusian Regional Government Finance (SAS PI-0458-2016)]. The work of MQ-V was supported by a predoctoral fellowship (BOE 22/10/2019) from the Spanish Ministry of Science, Innovation and Universities. This study is part of her doctoral research in the Biomedicine program at the University of Granada.

Mesenchymal stem cell-derived exosomesregenerative medicinemesenchymal stem cell-derived exosomesReviewimmunomodulationRegenerative medicineskin autoimmune diseasesImmunomodulationCell and Developmental Biologyexosomes-based therapyMedicineSkin wound healinglcsh:QH301-705.5Skin repairintegumentary systembusiness.industryRegeneration (biology)Mesenchymal stem cellCell BiologyExosomes-based therapyMicrovesicleslcsh:Biology (General)skin wound healingRegenerative medicineCancer researchSkin autoimmune diseasesWound healingbusinessDevelopmental BiologyFrontiers in Cell and Developmental Biology
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